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When the brakes are lost: LNK dysfunction in mice, men, and myeloproliferative neoplasms
Stephen T. Oh
Division of Hematology
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
Institute of Clinical and Translational Sciences (ICTS)
Siteman Cancer Center
Bursky Center for Human Immunology & Immunotherapy Programs (CHiiPs)
Department of Medicine
Rheumatic Diseases Research Resource-Based Center
DBBS - Molecular Cell Biology
DBBS - Immunology
DBBS - Molecular Genetics and Genomics
DBBS - Cancer Biology
Research output
:
Contribution to journal
›
Review article
›
peer-review
7
Scopus citations
Overview
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Keyphrases
Braking
100%
Myeloproliferative Neoplasms
100%
JAK-STAT Pathway
50%
Janus Kinase 2 (JAK2)
25%
STAT Activation
25%
Murine Model
12%
Tyrosine Kinase
12%
Targeted Therapy
12%
Adaptor Protein
12%
Regulatory Elements
12%
Negative Feedback Loop
12%
JAK-STAT
12%
JAK2 mutation
12%
Activating mutation
12%
Hyperproliferative
12%
MPL mutation
12%
Negative Feedback Regulation
12%
JAK2 Inhibitor
12%
Thrombopoietin Receptor
12%
Biochemistry, Genetics and Molecular Biology
Janus Kinase
100%
JAK-STAT Signaling
100%
JAK-STAT Signaling Pathway
75%
Tyrosine
25%
Genetics
25%
Regulatory Element
25%
Thrombopoietin Receptor
25%