Abstract
In this issue, Dang et al. (2011) report a role for the hypoxia-induced transcription factor HIF-1α in regulating the differentiation of T cell subsets that impact both inflammation and its resolution. Their findings illustrate how metabolic signals can alter the balance between inflammation and tolerance and present a potential therapeutic opportunity for the treatment of diseases associated with chronic inflammation.
Original language | English |
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Pages (from-to) | 673-674 |
Number of pages | 2 |
Journal | Cell |
Volume | 146 |
Issue number | 5 |
DOIs | |
State | Published - Sep 2 2011 |