Vancomycin-induced gut microbial dysbiosis alters enteric neuron–macrophage interactions during a critical period of postnatal development

Keyphrases

• Vancomycin 100%
• Postnatal Development 100%
• Gut Dysbiosis 100%
• Macrophages 30%
• Enteric Nervous System Development 30%
• Colonic Macrophages 30%
• Adult Mice 20%
• Enteric Nervous System 20%
• Antibiotic Exposure 20%
• Macrophage Populations 20%
• Early-life Antibiotics 20%
• Mouse Model 10%
• Gene-deficient Mice 10%
• Microbiome 10%
• Necrotizing Enterocolitis 10%
• CCR2 10%
• Yolk Sac 10%
• Broad-spectrum Antibiotics 10%
• Tissue-resident 10%
• Inflammatory Macrophages 10%
• Neuroimmune Interactions 10%
• Lineage Tracing 10%
• Dysbiosis 10%
• Lifesaving 10%
• Mouse Pups 10%
• Bone Marrow-derived Macrophages 10%
• Circulating Monocytes 10%
• Suspected Sepsis 10%
• Neuron Density 10%
• Neonatal Gut 10%
• Intestinal Macrophages 10%
• Single-cell RNA Sequencing (scRNA-seq) 10%
• Late-onset Sepsis 10%
• Muscularis Macrophages 10%
• Premature Neonates 10%

Immunology and Microbiology

• Macrophage 100%
• Postnatal Development 100%
• Dysbiosis 100%
• Intestine Innervation 40%
• Nervous System Development 30%
• Antimicrobial Agent 30%
• Mouse Model 10%
• Monocyte 10%
• Lineages 10%
• RNA Sequence 10%
• CCR2 10%
• Yolk Sac 10%
• Enterocolitis 10%
• Bone Marrow-Derived Macrophage 10%
• Microbiome 10%