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Uterine function in the mouse requires speckle-type poz protein
Lan Hai
, Maria M. Szwarc
, Bin He
, David M. Lonard
, Ramakrishna Kommagani
, Francesco J. DeMayo
, John P. Lydon
Division of Clinical Research
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
Institute of Clinical and Translational Sciences (ICTS)
Siteman Cancer Center
Research output
:
Contribution to journal
›
Article
›
peer-review
8
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Scopus citations
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Keyphrases
Uterus
100%
Uterine Function
100%
Mouse Model
50%
Endometrial Cancer
50%
Transcript Level
25%
Molecular Analysis
25%
Somatic mutation
25%
Oncogenic Drivers
25%
Cell Signaling
25%
Protein Level
25%
E3 Ubiquitin Ligase
25%
Cyst
25%
Gain-of-function mutation
25%
Proteome
25%
Cellular Processes
25%
Adaptor
25%
Transcriptional Regulation
25%
Expression Level
25%
Gynecologic Malignancies
25%
Genomic Analysis
25%
Endometrium
25%
Endometrial Decidualization
25%
Epithelial Ingrowth
25%
Protein Loss
25%
Chromatin Remodeling
25%
Estrogen Receptor 1 (ESR1)
25%
GATA2
25%
Etiopathogenesis
25%
Embryo Implantation
25%
Uterine Cells
25%
Fertility Studies
25%
Biochemistry, Genetics and Molecular Biology
Mouse Model
100%
Somatic Mutation
50%
Signal Transduction
50%
Wild Type
50%
Carcinogenesis
50%
Proteome
50%
Loss of Function Mutation
50%
GATA2
50%
Chromatin Remodeling
50%
Ubiquitin-Conjugating Enzyme
50%
Endometrium
50%
Decidualization
50%
Nidation
50%
Genomics
50%
Medicine and Dentistry
Cancer
100%
Somatic Mutation
50%
Ubiquitin Protein Ligase E3
50%
Decidualization
50%
Carcinogenesis
50%
Signal Transduction
50%
Loss of Function Mutation
50%
Endometrium
50%
Uterine Cancer
50%
Transcription Factor GATA 2
50%
Chromatin Remodeling
50%
Nidation
50%
Protein Depletion
50%
Proteome
50%