Type i IFNs regulate inflammation, vasculopathy, and fibrosis in chronic cutaneous graft-versus-host disease

Keyphrases

• Inflammation 100%
• Fibrosis 100%
• Type I IFN 100%
• Vasculopathy 100%
• Cutaneous Graft-versus-host Disease 100%
• IFNAR1 83%
• IFN-stimulated Genes 50%
• Gene Signature 33%
• Scleroderma 33%
• IFN Receptors 16%
• Interferon-α (IFN-α) 16%
• Overexpression 16%
• Graft-versus-host Disease (GvHD) 16%
• Immune Response 16%
• B Cells 16%
• T Cells 16%
• Autoimmunity 16%
• Viral Infection 16%
• Autoimmune Disease 16%
• Injury Response 16%
• Vascular Injury 16%
• Pathological Role 16%
• Plasmacytoid Dendritic Cells 16%
• Lymphocyte Survival 16%
• Neutralizing Abs 16%
• Dermal Fibrosis 16%
• Autoreactive Lymphocyte 16%
• Dermal Inflammation 16%
• Alloresponse 16%
• Skin Fibrosis 16%
• Monocyte Maturation 16%
• Sclerodermatous Graft-versus-host Disease 16%

Immunology and Microbiology

• Graft-Versus-Host Disease 100%
• Scleroderma 66%
• T Cell 33%
• Immune Response 33%
• Monocyte 33%
• Lymphocyte 33%
• B Cell 33%
• Autoimmunity 33%
• Viral Disease 33%
• Maturation 33%
• Autoimmune Disease 33%
• Dermatitis 33%
• Plasmacytoid Dendritic Cell 33%