Tubulointerstitial Diseases

J. P. Gaut; H. Liapis

doi:10.1016/B978-0-12-386456-7.05412-5

Tubulointerstitial Diseases

Research output: Chapter in Book/Report/Conference proceeding › Chapter › peer-review

Abstract

A wide variety of pathologic processes may affect the renal tubulointerstitium. The cLinical term acute kidney injury refers to a rapid increase in serum creatinine concentration. Under the majority of circumstances, such a rapid rise in serum creatinine is related to acute tubular damage. However, the etiologies of acute tubular damage are varied and may overlap, particularly in the critical care setting. This chapter discusses various causes of tubulointerstitial disease and their underlying pathophysiologic mechanisms.

Original language	English
Title of host publication	Pathobiology of Human Disease
Subtitle of host publication	A Dynamic Encyclopedia of Disease Mechanisms
Publisher	Elsevier Inc.
Pages	2853-2856
Number of pages	4
ISBN (Electronic)	9780123864567
ISBN (Print)	9780123864574
DOIs	https://doi.org/10.1016/B978-0-12-386456-7.05412-5
State	Published - Jan 1 2014

Keywords

Acute interstitial nephritis
Acute kidney injury
Acute pyelonephritis
Chronic pyelonephritis
Oxalate nephropathy
Sepsis
Uric acid nephropathy

Access to Document

10.1016/B978-0-12-386456-7.05412-5

Cite this

@inbook{8248ac158fde4653870204a6742767d8,

title = "Tubulointerstitial Diseases",

abstract = "A wide variety of pathologic processes may affect the renal tubulointerstitium. The cLinical term acute kidney injury refers to a rapid increase in serum creatinine concentration. Under the majority of circumstances, such a rapid rise in serum creatinine is related to acute tubular damage. However, the etiologies of acute tubular damage are varied and may overlap, particularly in the critical care setting. This chapter discusses various causes of tubulointerstitial disease and their underlying pathophysiologic mechanisms.",

keywords = "Acute interstitial nephritis, Acute kidney injury, Acute pyelonephritis, Chronic pyelonephritis, Oxalate nephropathy, Sepsis, Uric acid nephropathy",

author = "Gaut, {J. P.} and H. Liapis",

year = "2014",

month = jan,

day = "1",

doi = "10.1016/B978-0-12-386456-7.05412-5",

language = "English",

isbn = "9780123864574",

pages = "2853--2856",

booktitle = "Pathobiology of Human Disease",

publisher = "Elsevier Inc.",

}

TY - CHAP

T1 - Tubulointerstitial Diseases

AU - Gaut, J. P.

AU - Liapis, H.

PY - 2014/1/1

Y1 - 2014/1/1

N2 - A wide variety of pathologic processes may affect the renal tubulointerstitium. The cLinical term acute kidney injury refers to a rapid increase in serum creatinine concentration. Under the majority of circumstances, such a rapid rise in serum creatinine is related to acute tubular damage. However, the etiologies of acute tubular damage are varied and may overlap, particularly in the critical care setting. This chapter discusses various causes of tubulointerstitial disease and their underlying pathophysiologic mechanisms.

AB - A wide variety of pathologic processes may affect the renal tubulointerstitium. The cLinical term acute kidney injury refers to a rapid increase in serum creatinine concentration. Under the majority of circumstances, such a rapid rise in serum creatinine is related to acute tubular damage. However, the etiologies of acute tubular damage are varied and may overlap, particularly in the critical care setting. This chapter discusses various causes of tubulointerstitial disease and their underlying pathophysiologic mechanisms.

KW - Acute interstitial nephritis

KW - Acute kidney injury

KW - Acute pyelonephritis

KW - Chronic pyelonephritis

KW - Oxalate nephropathy

KW - Sepsis

KW - Uric acid nephropathy

UR - http://www.scopus.com/inward/record.url?scp=85043287125&partnerID=8YFLogxK

U2 - 10.1016/B978-0-12-386456-7.05412-5

DO - 10.1016/B978-0-12-386456-7.05412-5

M3 - Chapter

AN - SCOPUS:85043287125

SN - 9780123864574

SP - 2853

EP - 2856

BT - Pathobiology of Human Disease

PB - Elsevier Inc.

ER -

Tubulointerstitial Diseases

Abstract

Keywords

Access to Document

Other files and links

Fingerprint

Cite this