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TREM2 variants: New keys to decipher Alzheimer disease pathogenesis
Marco Colonna
, Yaming Wang
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
Institute of Clinical and Translational Sciences (ICTS)
Siteman Cancer Center
Division of Immunobiology
Bursky Center for Human Immunology & Immunotherapy Programs (CHiiPs)
Center for Brain Immunology & Glia (BIG)
Rheumatic Diseases Research Resource-Based Center
DBBS - Immunology
DBBS - Molecular Microbiology and Microbial Pathogenesis
Hope Center for Neurological Disorders
Research output
:
Contribution to journal
›
Article
›
peer-review
295
Scopus citations
Overview
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Keyphrases
Alzheimer's Disease
16%
Alzheimer's Disease Pathology
100%
Brain Microglia
16%
Brain Response
16%
Cell Surface
16%
Familial Alzheimer's Disease
16%
Gene Variants
16%
Genome-wide Association Study
16%
Immune Signaling
16%
Microglia
16%
Microglial Response
16%
Microglial Senescence
16%
New Key
100%
Non-family
16%
Plaque
16%
Rare Variants
16%
Risk Factors
16%
Triggering Receptor Expressed on Myeloid Cells 2 (TREM2)
100%
Medicine and Dentistry
Alzheimer's Disease
100%
Amyloid Protein
16%
Cell Surface
16%
Familial Alzheimer's Disease
16%
Genome Wide Association Study
16%
Immune System Receptors
16%
Microglia
33%
Triggering Receptor Expressed on Myeloid Cells 2
100%