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Toll-like receptors and IFN-α: Partners in autoimmunity
Marco Colonna
Division of Immunobiology
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
Institute of Clinical and Translational Sciences (ICTS)
Siteman Cancer Center
Bursky Center for Human Immunology & Immunotherapy Programs (CHiiPs)
Center for Brain Immunology & Glia (BIG)
Rheumatic Diseases Research Resource-Based Center
DBBS - Immunology
DBBS - Molecular Microbiology and Microbial Pathogenesis
Hope Center for Neurological Disorders
Research output
:
Contribution to journal
›
Review article
›
peer-review
26
Scopus citations
Overview
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Keyphrases
Toll-like
100%
Autoimmunity
100%
Viral Infection
100%
Mouse Model
50%
Antigen-presenting Cells
50%
CD8+ T Cells
50%
Autoimmune Disease
50%
Chemokines
50%
Activated Cells
50%
Liver Damage
50%
Self-reactivity
50%
Autoreactive T Cells
50%
Hepatitis-viral
50%
TLR Stimulation
50%
Autoimmune Hepatitis
50%
Immunology and Microbiology
Autoimmunity
100%
Viral Disease
100%
Toll-Like Receptor
100%
Mouse Model
50%
T Cell
50%
Tumor Necrosis Factor
50%
Antigen-Presenting Cell
50%
Cytotoxic T-Cell
50%
Autoimmune Disease
50%
Chemokine
50%
Autoimmune Hepatitis
50%