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The tyrosine phosphatase SHP-1 dampens murine Th17 development
Ileana S. Mauldin
, Kenneth S. Tung
,
Ulrike M. Lorenz
Division of Laboratory and Genomic Medicine
Bursky Center for Human Immunology and Immunotherapy
Research output
:
Contribution to journal
›
Article
›
peer-review
26
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Scopus citations
Overview
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Dive into the research topics of 'The tyrosine phosphatase SHP-1 dampens murine Th17 development'. Together they form a unique fingerprint.
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Immunology and Microbiology
Cytokine
100%
STAT3
100%
Interleukin 6
100%
Th17 Cell
100%
Tyrosine
100%
Phosphatase
100%
T Cell
50%
T-Helper Cell
50%
Transgenics
50%
Gene Deletion
50%
CD4
50%
Autoimmune Disease
50%
Proinflammatory Cytokine
50%
T Helper Cell
50%
Pathogenic Fungi
50%
Interleukin 17
50%
Biochemistry, Genetics and Molecular Biology
Cytokine
100%
T Cell
100%
Protein Tyrosine Phosphatase
100%
CD4
100%
Interleukin 6
100%
STAT3
100%
Genetics
50%
Transgenics
50%
Gene Deletion
50%
Autoimmunity
50%
T Helper Cell
50%
Proinflammatory Cytokine
50%
Interleukin 17
50%
Keyphrases
T Helper 17 (Th17)
100%
Protein Tyrosine Phosphatase 1B (PTP1B)
100%
SHP-1
100%
Interleukin-6
20%
Interleukin-21 (IL-21)
20%
Genomic Deletion
10%
Inflammatory Cytokines
10%
Phosphorylation
10%
T Cells
10%
Interleukin-17 (IL-17)
10%
Hyper-response
10%
Pharmacologic
10%
Autoimmune Disease
10%
Dominant Negative
10%
Pharmacological Inhibitors
10%
CD4+ T Helper Cells
10%
Pathogenic Bacteria
10%
New Drug Targets
10%
Pathogenic Role
10%
Transgenic Expression
10%
Pathogenic Fungi
10%
Complementary Approach
10%
STAT3 Signaling
10%
Beneficial Role
10%
Th17 Cell Differentiation
10%
Primary CD4+ T Cells
10%
Pharmacology, Toxicology and Pharmaceutical Science
Cytokine
100%
Protein Tyrosine Phosphatase
100%
Protein Tyrosine Phosphatase SHP 1
100%
Interleukin 6
66%
Autoimmune Disease
33%
Interleukin 17
33%