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The PPAR regulatory system in cardiac physiology and disease
Brian N. Finck
Division of Nutritional Science & Obesity Medicine
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
Institute of Clinical and Translational Sciences (ICTS)
Bursky Center for Human Immunology & Immunotherapy Programs (CHiiPs)
DBBS - Molecular Cell Biology
Research output
:
Contribution to journal
›
Review article
›
peer-review
182
Scopus citations
Overview
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Keyphrases
Cardiac Disease
100%
Cardiac Function
50%
Cardiac Metabolism
50%
Cardiac Physiology
100%
Cardiac Structure
50%
Coactivator
50%
Deactivation
50%
Fatty Acid Utilization
50%
Glucose Utilization
50%
Metabolic Disease
50%
Metabolism
100%
Myocardial Energy Metabolism
50%
Myocardial Metabolism
50%
Nuclear Receptor
50%
Peroxisome Proliferator-activated Receptor
50%
PGC-1
50%
Pharmacological Therapy
50%
Potential Impact
50%
PPAR Activation
50%
Regulatory System
100%
Therapeutic Avenues
50%
Transcription Factor
50%
Medicine and Dentistry
Cardiac Physiology
100%
Cell Nucleus Receptor
20%
Energy Metabolism
20%
Fatty Acid
20%
Glucose Utilization
20%
Heart Disease
100%
Heart Disease
20%
Heart Muscle Metabolism
40%
Metabolic Disorder
20%
Peroxisome Proliferator Activated Receptor
100%
Receptor Family
20%
Transcription Factors
20%
Pharmacology, Toxicology and Pharmaceutical Science
Cell Nucleus Receptor
20%
Diseases
100%
Fatty Acid
20%
Heart Disease
20%
Peroxisome Proliferator Activated Receptor
100%
Transcription Factors
20%