The decay accelerating factor mutation I197V found in hemolytic uraemic syndrome does not impair complement regulation

Keyphrases

• Acute Renal Failure 14%
• Alternative Pathway 14%
• Atypical Hemolytic Uremic Syndrome (aHUS) 100%
• Atypical HUS 100%
• B Lymphocytes 14%
• CD46 14%
• CD55 14%
• Classical Pathway 14%
• Clinical Triad 14%
• Cofactor Activity 28%
• Complement Regulation 100%
• Complement Regulatory Proteins 14%
• Complement System 14%
• Convertase 14%
• Decay Accelerating Factor 100%
• Gene Cluster 14%
• Healthy Controls 14%
• Membrane Cofactor Protein 14%
• Microangiopathic Hemolytic Anemia 14%
• Molecular Modeling 14%
• Non-associated 14%
• Producing Escherichia Coli 14%
• Protein Factor 14%
• Regulators of Complement Activation 28%
• Shiga Toxin 14%
• Thrombocytopenia 14%

Biochemistry, Genetics and Molecular Biology

• B Cell 16%
• CD46 33%
• Complement Activation 33%
• Complement System 16%
• Convertase 16%
• Decay-Accelerating Factor 100%
• Epstein Barr Virus 16%
• Escherichia coli 16%
• Factor H 16%
• Fibrinogen 16%
• Gene Cluster 16%
• Gene Linkage 33%
• Molecular Model 16%
• Shiga-Like Toxin 16%
• Thrombocytopenia 16%
• Thrombotic Thrombocytopenic Purpura 16%
• Wild Type 33%

Immunology and Microbiology

• B Cell 16%
• CD46 33%
• Complement Activation 33%
• Complement System 16%
• Decay-Accelerating Factor 100%
• Epstein Barr Virus 16%
• Escherichia coli 16%
• Factor H 16%
• Fibrinogen 16%
• Gene Cluster 16%
• Molecular Model 16%
• Shiga-Like Toxin 16%
• Thrombotic Thrombocytopenic Purpura 16%
• Wild Type 33%