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Th17 cells can provide B cell help in autoantibody induced arthritis
Cynthia L. Hickman-Brecks
, Jennifer L. Racz
, Debra M. Meyer
, Timothy P. LaBranche
, Paul M. Allen
Institute of Clinical and Translational Sciences (ICTS)
Siteman Cancer Center
Division of Immunobiology
Bursky Center for Human Immunology & Immunotherapy Programs (CHiiPs)
Research output
:
Contribution to journal
›
Article
›
peer-review
57
Scopus citations
Overview
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Keyphrases
Arthritis
100%
T Helper 17 (Th17)
100%
Autoantibodies
100%
B Cell Help
100%
T Cells
33%
Interleukin-17 (IL-17)
33%
T-bet
33%
CD4+ T Cells
22%
H-2B
22%
Glucose-6-phosphatase (G6Pase)
22%
T-cell Transfer
22%
Disease Severity
11%
Gene-deficient Mice
11%
IgG2b
11%
Early Disease
11%
Popliteal Lymph Node
11%
Cytokine Expression
11%
T-cell Mediated
11%
Ankle
11%
Disease Development
11%
Cell Transfer
11%
IgG1
11%
Antibody Production
11%
Chronic Arthritis
11%
Destructive Arthritis
11%
Immunology and Microbiology
B Cell
100%
Autoantibodies
100%
Arthritis
100%
Th17 Cell
100%
T Cell
66%
T-Helper Cell
22%
T Cell Receptor
22%
Neutralization
22%
Glucose 6 Phosphate Isomerase
22%
Cytokine
11%
Immunoglobulin G2b
11%
Popliteal Lymph Node
11%
Immunoglobulin G1
11%
Transfer Cell
11%
Ankle
11%
Antibody Generation
11%