Abstract
After damage to the endothelium, platelets prevent excessive bleeding by adhering to the exposed ECM. In this issue of Blood, Kim and colleagues1 describe a novel function of TGFBIp that potentiates platelet adhesion and activation, a protein with a previously established role in corneal dystrophies.
Original language | English |
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Pages (from-to) | 5113-5114 |
Number of pages | 2 |
Journal | Blood |
Volume | 114 |
Issue number | 25 |
DOIs |
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State | Published - Dec 10 2009 |