Targeted deletion of the cytosolic Cu/Zn-superoxide dismutase gene (Sod1) increases susceptibility to noise-induced hearing loss

Keyphrases

• Superoxide Dismutase 1 (SOD1) 100%
• Targeted Deletion 100%
• Noise-induced Hearing Loss 100%
• Superoxide Dismutase Genes 100%
• Zn-superoxide Dismutase 100%
• Reactive Oxygen Species 80%
• Temporary Threshold Shift 60%
• Knock-in Mouse Model 40%
• Cochlea 40%
• Noise Effects 40%
• Superoxide Ion 40%
• Cell Behavior 20%
• Vulnerability 20%
• Cell Death 20%
• Disease States 20%
• Cell Components 20%
• Superoxide 20%
• Chronic Disease 20%
• Noise Exposure 20%
• Broadband Noise 20%
• Superoxide Dismutase 20%
• Acute Injury 20%
• Reactive Oxygen Species Level 20%
• Auditory Brainstem Response 20%
• Central Nervous System Injury 20%
• Cellular Metabolism 20%
• Antioxidant Enzymes 20%
• Unlinked 20%
• Hydroxyl Radical 20%
• Hearing Sensitivity 20%
• Loud Noise 20%
• Threshold Height 20%
• Three-mode 20%
• Reactive Oxygen Species Regulation 20%

Neuroscience

• Copper Zinc Superoxide Dismutase 100%
• Reactive Oxygen Species 100%
• Central Nervous System 16%
• Metabolic Pathway 16%
• Cell Function 16%
• Cochlea 16%
• Sensation of Hearing 16%
• Auditory Brainstem Response 16%
• Cell Damage 16%
• Antioxidant 16%
• Superoxide Dismutase 16%