STAT3 gain-of-function mutations connect leukemia with autoimmune disease by pathological NKG2D^hi CD8⁺ T cell dysregulation and accumulation

Etienne Masle-Farquhar, Katherine J.L. Jackson, Timothy J. Peters, Ghamdan Al-Eryani, Mandeep Singh, Kathryn J. Payne, Geetha Rao, Danielle T. Avery, Gabrielle Apps, Jennifer Kingham, Christopher J. Jara, Ksenia Skvortsova, Alexander Swarbrick, Cindy S. Ma, Daniel Suan, Gulbu Uzel, Ignatius Chua, Jennifer W. Leiding, Kaarina Heiskanen, Kahn PreeceLeena Kainulainen, Michael O'Sullivan, Megan A. Cooper, Mikko R.J. Seppänen, Satu Mustjoki, Shannon Brothers, Tiphanie P. Vogel, Robert Brink, Stuart G. Tangye, Joanne H. Reed, Christopher C. Goodnow

Division of Rheumatology
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
DBBS - Human and Statistical Genetics
DBBS - Immunology
Institute of Clinical and Translational Sciences
Bursky Center for Human Immunology & Immunotherapy Programs (CHiiPs)
Rheumatic Diseases Research Resource-Based Center
Siteman Cancer Center
COVID-19 Researchers

Research output: Contribution to journal › Article › peer-review

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STAT3 gain-of-function mutations connect leukemia with autoimmune disease by pathological NKG2D^hi CD8⁺ T cell dysregulation and accumulation

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Medicine & Life Sciences

STAT3 gain-of-function mutations connect leukemia with autoimmune disease by pathological NKG2Dhi CD8+ T cell dysregulation and accumulation

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Medicine & Life Sciences

STAT3 gain-of-function mutations connect leukemia with autoimmune disease by pathological NKG2D^hi CD8⁺ T cell dysregulation and accumulation