Splicing factor 3B subunit 1 interacts with HIV tat and plays a role in viral transcription and reactivation from latency

Keyphrases

• Viral Reactivation 100%
• Reactivation from Latency 100%
• HIV-1 Tat 100%
• Viral Transcription 100%
• Splicing Factor 3B Subunit 1 100%
• HIV Transcription 33%
• HIV Reactivation 25%
• Chromatin 16%
• Virus 16%
• Viral Proteins 16%
• HIV Cure 16%
• Remission 8%
• Knockdown 8%
• Provirus 8%
• HIV-1 Replication 8%
• Resting CD4+ T Cells 8%
• Therapeutic Target 8%
• T Cells 8%
• Malignant Cells 8%
• Current Therapies 8%
• Viral Genome 8%
• Splice Site 8%
• Pharmacological Inhibitors 8%
• Cellular Factors 8%
• HIV RNA 8%
• Viral Persistence 8%
• Viral Replication 8%
• Host Targets 8%
• Gene Transcript 8%
• Viral Reservoir 8%
• Latency-reversing Agents 8%
• RNA Polymerase II (RNAPII) 8%
• Sustained Remission 8%
• Splicing Factor 8%
• Block-and-lock 8%
• HIV Remission 8%
• HIV Dependency Factors 8%

Immunology and Microbiology

• Human Immunodeficiency Virus 100%
• Viral Transcription 100%
• Tat (HIV) 100%
• Viral Protein 16%
• Herpes Simplex Virus Protein Vmw65 16%
• Virus 16%
• T Cell 8%
• Provirus 8%
• T-Helper Cell 8%
• Promoter Region 8%
• Virus Genome 8%
• Viral Replication 8%
• RNA Polymerase 8%

Medicine and Dentistry

• Human Immunodeficiency Virus 100%
• Viral Transcription 100%
• Transactivator Protein 15%
• Viral Protein 15%
• Virus 15%
• Malignant Neoplasm 7%
• Provirus 7%
• In Vitro 7%
• T-Helper Cell 7%
• Promoter Region 7%
• T Cell 7%
• Virus Replication 7%
• RNA Polymerase II 7%
• Virus Genome 7%