Abstract

Although it was discovered more than 70 years ago that caloric restriction (CR) extends the life span of rodents, the molecular basis of CR has remained obscure. Recently, NAD-dependent Sir2 deacetylases have emerged as key regulators that mediate the adaptive response to CR. Here, we describe the tissue-dependent functions of the mammalian Sir2 ortholog, Sirt1/Sir2α and present a molecular framework in which Sirt1 orchestrates multiple physiological changes into a coordinated response to CR in mammals.

Original languageEnglish
Pages (from-to)11-17
Number of pages7
JournalDrug Discovery Today: Disease Mechanisms
Volume3
Issue number1
DOIs
StatePublished - 2006

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