SERPING1 mRNA overexpression in monocytes from HIV+ patients

C. Sanfilippo, D. Cambria, A. Longo, M. Palumbo, R. Avola, M. Pinzone, G. Nunnari, F. Condorelli, G. Musumeci, R. Imbesi, P. Castogiovanni, L. Malaguarnera, Michelino Di Rosa

Research output: Contribution to journalArticlepeer-review

13 Scopus citations

Abstract

Objective: The HIV-1 virus activates the complement system, an essential element of the immune system. SERPING1 is a protease inhibitor that disables C1r/C1s in the C1 complex of the classical complement pathway. Methods: In this paper, we performed an analysis of several microarrays deposited in GEO dataset to demonstrate that SERPING1 mRNA is modulated in CD14+ monocytes from HIV-1-infected individuals. In addition, data were validated on monocytes isolated from seronegative healthy volunteers, treated with IFNs. Results: Our analysis shows that SERPING1 mRNA is overexpressed in monocytes from HIV-1+ patients and the expression levels correlate positively with viral load and negatively with the CD4+ T-cell count. Of note, anti-retroviral therapy is able to reduce the levels of SERPING1 mRNA, ex vivo. In addition, we found that 30% of the SERPING1 genes network is upregulated in monocytes from HIV-1+ patients. Noteworthy, the expression levels of IFITM1—an antiviral molecule belonging to the genes network—correlate positively with SERPING1 expression. Interestingly, the monocytes treatment with IFN-gamma, IFN-beta and IFN-alpha significantly upregulates the SERPING1 mRNA expression levels. Conclusions: From the outcome of our investigation, it is possible to conclude that SERPING1 and its network serve as important components of the innate immune system to restrict HIV-1 infection.

Original languageEnglish
Pages (from-to)1107-1116
Number of pages10
JournalInflammation Research
Volume66
Issue number12
DOIs
StatePublished - Dec 1 2017

Keywords

  • C1-INH
  • Complement
  • HIV-1
  • IFN-gamma
  • Monocyte
  • SERPING1

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