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Senescent glia spell trouble in Alzheimer’s disease
David Holtzman
,
Jason Ulrich
Department of Neurology
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
Institute of Clinical and Translational Sciences (ICTS)
Bursky Center for Human Immunology & Immunotherapy Programs (CHiiPs)
Center for Brain Immunology & Glia (BIG)
Hope Center for Neurological Disorders
DBBS - Molecular Cell Biology
DBBS - Neurosciences
Intellectual and Developmental Disabilities Research Center (IDDRC)
Research output
:
Contribution to journal
›
Article
›
peer-review
25
Scopus citations
Overview
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Medicine and Dentistry
DNA Damage
100%
Alzheimer's Disease
100%
Amyloid Plaque
100%
Oligodendrocyte
100%
Progenitor Cell
100%
Tissue Injury
100%
Cell Stress
100%
Glia
100%
Cell DNA
100%
Pathology of Alzheimer's Disease
100%
Diseases
100%
Keyphrases
Alzheimer's Disease
100%
Glia
100%
Senescence
100%
Amyloid Plaques
50%
Aging
50%
Alzheimer's Disease Pathology
50%
Age-associated
50%
Cellular Stress
50%
Tissue Damage
50%
Tissue Inflammation
50%
Oligodendrocyte Precursor Cells
50%
Tissue Disease
50%
DNA Damage Stress
50%
Senescent Cells
50%
Biochemistry, Genetics and Molecular Biology
DNA Damage
100%
Progenitor Cell
100%
Cell Stress
100%
Oligodendrocyte Progenitor
100%
Amyloid Protein
100%