Abstract

In this issue of Immunity, Xu et al. reveal that dermal dendritic cells produce interleukin-31, which acts on neurons to promote wound itch. Their findings link itch associated with deeper wounds—wounds that extend beyond the epithelium—to the cells and cytokines that mediate wound healing.

Original languageEnglish
Pages (from-to)235-237
Number of pages3
JournalImmunity
Volume53
Issue number2
DOIs
StatePublished - Aug 18 2020

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