TY - JOUR
T1 - ScFv-mediated in vivo targeting of DAF to erythrocytes inhibits lysis by complement
AU - Spitzer, Dirk
AU - Unsinger, Jacqueline
AU - Bessler, Monica
AU - Atkinson, John P.
N1 - Funding Information:
We thank L. Jerabek and I. Weissman from Stanford University (Stanford University, School of Medicine, Stanford, California, USA) for providing the hybridoma cell line TER-119, T. Böldicke and H. Weich from the German Research Center for Biotechnology (GBF, Braunschweig, Germany) for providing the scFv-KDR cDNA (clone A7), Deborah LaFlame for her assistance in the mouse colony, Dennis Hourcade for helpful suggestions and Curtis Parvin for statistical advice. This work was supported by 5 RO1 AI37618 (J.P.A.), 5 RO1 AT41592 (J.P.A.), HL63208 (M.B.), CA89091 (M.B.), and the American Heart Association grant 0240037N (M.B.).
PY - 2004/2
Y1 - 2004/2
N2 - Antibodies or antibody fragments represent a powerful class of targeting moieties to specifically attach proteins to the surface of a given cell or tissue. Since the presence of constant antibody domains in these targeted fusion proteins may have certain disadvantages, we report here the reduction of the targeting epitope to the variable regions of an Ab by the generation of a single chain antibody fragment (scFv). As an effector molecule, we attached the human complement regulatory protein (CRP) decay-accelerating factor (DAF) at its amino-terminus with a scFv specific for TER-119, a red blood cell (RBC) restricted surface antigen of the mouse. This heterologous system enabled us to study (a) the applicability of a scFv as a targeting domain, (b) the functionality of the effector molecule with respect to regulation of the complement cascade in vitro, and (c) the in vivo biodistribution characteristics of a scFv-DAF fusion protein attached to a clinically relevant target cell type. RBCs from C57BL/6 mice loaded in vitro or in vivo with this fusion protein were significantly protected against lysis by human complement. After intravenous injection, a homogeneous population of in vivo tagged RBCs was maintained throughout a 6-day follow-up. This result and in vitro mixing experiments indicated that there was an equilibration of the fusion protein between tagged and non-tagged RBCs. Thus, scFv-mediated targeting of proteins to a selected cell or tissue surface has promise as a means to supplement absent or defective plasma membrane constituents. This approach should therefore be applicable for diseases caused by a membrane protein deficiency such as paroxysmal nocturnal hemoglobinuria (PNH).
AB - Antibodies or antibody fragments represent a powerful class of targeting moieties to specifically attach proteins to the surface of a given cell or tissue. Since the presence of constant antibody domains in these targeted fusion proteins may have certain disadvantages, we report here the reduction of the targeting epitope to the variable regions of an Ab by the generation of a single chain antibody fragment (scFv). As an effector molecule, we attached the human complement regulatory protein (CRP) decay-accelerating factor (DAF) at its amino-terminus with a scFv specific for TER-119, a red blood cell (RBC) restricted surface antigen of the mouse. This heterologous system enabled us to study (a) the applicability of a scFv as a targeting domain, (b) the functionality of the effector molecule with respect to regulation of the complement cascade in vitro, and (c) the in vivo biodistribution characteristics of a scFv-DAF fusion protein attached to a clinically relevant target cell type. RBCs from C57BL/6 mice loaded in vitro or in vivo with this fusion protein were significantly protected against lysis by human complement. After intravenous injection, a homogeneous population of in vivo tagged RBCs was maintained throughout a 6-day follow-up. This result and in vitro mixing experiments indicated that there was an equilibration of the fusion protein between tagged and non-tagged RBCs. Thus, scFv-mediated targeting of proteins to a selected cell or tissue surface has promise as a means to supplement absent or defective plasma membrane constituents. This approach should therefore be applicable for diseases caused by a membrane protein deficiency such as paroxysmal nocturnal hemoglobinuria (PNH).
KW - Decay-accelerating factor
KW - Erythrocytes
KW - Fusion protein
KW - In vivo
KW - Single chain Fv
KW - Targeting
UR - https://www.scopus.com/pages/publications/0346023953
U2 - 10.1016/j.molimm.2003.10.017
DO - 10.1016/j.molimm.2003.10.017
M3 - Article
C2 - 14725787
AN - SCOPUS:0346023953
SN - 0161-5890
VL - 40
SP - 911
EP - 919
JO - Molecular Immunology
JF - Molecular Immunology
IS - 13
ER -