Resveratrol inhibits high glucose-induced PI3K/Akt/ERK-dependent interleukin-17 expression in primary mouse cardiac fibroblasts

Keyphrases

• Interleukin-17 (IL-17) 100%
• Cardiac Fibroblasts 100%
• High Glucose 100%
• AKT1 100%
• Resveratrol 100%
• Interleukin 25%
• Adenoviral 16%
• D-glucose 16%
• Collagen Production 16%
• Inflammatory Cytokines 8%
• Hyperglycemia 8%
• MRNA Expression 8%
• Phosphoinositide 3-kinase (PI3K) 8%
• Dominant Negative 8%
• Myocardial Injury 8%
• Therapeutic Potential 8%
• Low Glucose 8%
• Paracrine Mechanism 8%
• Basal Level 8%
• Mannitol 8%
• Post-transcriptional Mechanisms 8%
• Inflammation Injury 8%
• Myocardial Inflammation 8%
• Transcriptional Mechanism 8%
• Interleukin-17 Receptor 8%
• PD98059 8%
• Myocardial Remodeling 8%
• Amplification Loop 8%
• PI3K-p85 8%
• Phytoalexin 8%

Biochemistry, Genetics and Molecular Biology

• Interleukin 17 100%
• Protein Kinase B 100%
• Resveratrol 100%
• Fibroblast 100%
• Cytokine 25%
• Secretion (Process) 8%
• Hyperglycemia 8%
• Phosphoinositide 3-Kinase 8%
• Proinflammatory Cytokine 8%
• Interleukin-17 Receptor 8%
• Phytoalexin 8%
• Messenger RNA 8%
• Mannitol 8%

Immunology and Microbiology

• Fibroblast 100%
• Interleukin 17 100%
• Cytokine 25%
• Secretion (Process) 8%
• Proinflammatory Cytokine 8%
• Myocarditis 8%
• Interleukin-17 Receptor 8%