Reduced striatal dopamine underlies the attention system dysfunction in neurofibromatosis-1 mutant mice

Keyphrases

• Striatal Dopamine 100%
• Neurofibromatosis Type 1 (NF-1) 100%
• Mutant Mice 100%
• Attention System 100%
• Optic Pathway Glioma 80%
• Striatum 40%
• Dopaminergic Neurons 40%
• Methylphenidate 40%
• Genetically Engineered Mice 40%
• Rate Limiting 20%
• Cellular Dysfunction 20%
• Behavioral Abnormalities 20%
• Homeostasis 20%
• Non-selective 20%
• Catecholamines 20%
• Hyperactivity 20%
• Non-cell Autonomous 20%
• Cognitive Deficits 20%
• Substantia Nigra 20%
• Striatal 20%
• Selective Attention 20%
• Molecular Defects 20%
• Cell Projection 20%
• Neurite Extension 20%
• Dopaminergic Neuron Loss 20%
• L-DOPA 20%
• Striatal Dopamine Level 20%
• Clinical Problems 20%
• Mouse in Vivo 20%
• Dopamine Homeostasis 20%
• Environmental Stimuli 20%
• Genetically Engineered Mouse Models 20%
• Tyrosine Hydroxylase 20%
• Neuron Growth 20%
• Dopamine Level 20%
• Inherited Cancer Syndrome 20%
• Growth Cone 20%
• Dopamine Synthesis 20%
• Novel Object 20%

Neuroscience

• Neurofibromatosis Type 1 100%
• Dopaminergic 40%
• Striatum 40%
• Methylphenidate 40%
• Cognitive Disorders 20%
• In Vivo 20%
• Cell Loss 20%
• In Vitro 20%
• Neurite 20%
• Catecholamine 20%
• Hyperactivity 20%
• Substantia nigra 20%
• Growth Cone 20%
• Dopaminergic Neuron 20%
• Tyrosine Hydroxylase 20%