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Protein kinase cascades in the regulation of cardiac hypertrophy
Gerald W. Dorn
, Thomas Force
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
Institute of Clinical and Translational Sciences (ICTS)
Department of Medicine
DBBS - Molecular Cell Biology
DBBS - Human and Statistical Genetics
DBBS - Molecular Genetics and Genomics
Hope Center for Neurological Disorders
Research output
:
Contribution to journal
›
Review article
›
peer-review
568
Scopus citations
Overview
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Keyphrases
Protein Kinase
100%
Cardiac Development
100%
Cardiac Hypertrophy
100%
Kinase Cascade
100%
Phospholipase C
50%
Pathologic
50%
Molecular Targets
50%
Growth Hormone
50%
Paracrine
50%
Am(III)
50%
Physiologic Hypertrophy
50%
Cytosolic Calcium
50%
Exercise-induced
50%
PI3K-Akt Pathway
50%
Neurohormonal
50%
Human Condition
50%
Normal Growth
50%
Biomechanical Stress
50%
IGF Axis
50%
Physical Conditioning
50%
Biochemistry, Genetics and Molecular Biology
Protein Kinases
100%
Conditioning
100%
Animal Model
100%
Bovine Somatotropin
100%
Growth Hormone
100%
Lecithinase
100%
Protein Kinase B
100%
Medicine and Dentistry
Ventricular Hypertrophy
100%
Protein Kinases
100%
Isotopes of Calcium
50%
Conditioning
50%
Kinase
50%
Growth Hormone
50%
Lecithinase
50%
Protein Kinase B
50%
Pharmacology, Toxicology and Pharmaceutical Science
Heart Ventricle Hypertrophy
100%
Protein Kinases
100%
Molecular Target
50%
Growth Hormone
50%
Lecithinase
50%