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Potentiation of cAMP responses by metabotropic glutamate receptors depresses excitatory synaptic transmission by a kinase-independent mechanism
Robert W. Gereau IV
, P. Jeffrey Conn
Washington University Pain Center
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
Institute of Clinical and Translational Sciences (ICTS)
Department of Anesthesiology
Division of Pain Research
DBBS - Molecular Cell Biology
DBBS - Neurosciences
Research output
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Contribution to journal
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Article
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peer-review
69
Scopus citations
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Dive into the research topics of 'Potentiation of cAMP responses by metabotropic glutamate receptors depresses excitatory synaptic transmission by a kinase-independent mechanism'. Together they form a unique fingerprint.
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Keyphrases
Potentiation
100%
Excitatory Synaptic Transmission
100%
CAMP-dependent Protein Kinase
100%
Adrenergic Receptors
100%
Metabotropic Glutamate Receptor
100%
Co-activation
100%
Kinase-independent
100%
Hippocampus
50%
Metabolism
50%
Adenosine
50%
Synapse
50%
Extracellular Space
50%
Protein Kinase Activity
50%
5'-AMP
50%
Hippocampal Formation
50%
Rat Hippocampus
50%
Schaffer Collateral
50%
Adenosine Receptors
50%
Acute Depression
50%
Hormone-like Effects
50%
CAMP Level
50%
Neuroscience
Glutamate Receptor
100%
Cyclic Adenosine Monophosphate
100%
Synaptic Transmission
100%
Protein Kinase A
100%
Protein Kinases
60%
Hippocampus
40%
Adrenergic Receptor
40%
Metabolic Pathway
20%
Synapse
20%
Adenosine
20%
Adenosine Monophosphate
20%
Adenosine Receptor
20%
Hippocampal Formation
20%
Schaffer Collateral
20%
Local Hormone
20%