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Pim3 negatively regulates glucose-stimulated insulin secretion
Gregory Vlacich
, Martijn C. Nawijn
, Gene C. Webb
, Donald F. Steiner
Institute of Clinical and Translational Sciences (ICTS)
Siteman Cancer Center
Division of Clinical Research
Research output
:
Contribution to journal
›
Article
›
peer-review
14
Scopus citations
Overview
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Keyphrases
Negatively Regulate
100%
Glucose-stimulated Insulin Secretion
100%
PIM3
100%
ERK2
30%
SOCS6
30%
Insulin Sensitivity
15%
Cell Response
15%
Islet
15%
Glucose Stimulation
15%
Glucose-responsive
15%
Responsive Genes
15%
MIN6 Cells
15%
Phosphorylation
7%
Glucose Homeostasis
7%
Glucose Tolerance
7%
Overexpression
7%
Signaling Pathway
7%
Pancreatic Cells
7%
Insulin Secretion
7%
Pancreas
7%
Microarray Analysis
7%
Insulinoma
7%
Isolated Islets
7%
Global Function
7%
Regulation of Cell Functions
7%
Immunology and Microbiology
Insulin Release
100%
Insulin Sensitivity
66%
Secretion (Process)
33%
Cell Function
33%
Pancreas
33%
Dynamics
33%
Glucose Homeostasis
33%
Biochemistry, Genetics and Molecular Biology
Insulin Release
100%
Insulin Sensitivity
66%
Secretion (Process)
33%
Glucose Homeostasis
33%
Dynamics
33%
Cell Function
33%
Protein Kinase A
33%