Phase I trial of ATR inhibitor elimusertib with FOLFIRI in advanced or metastatic gastrointestinal malignancies (ETCTN 10406)

Anuradha Krishnamurthy, Hong Wang, John C. Rhee, Diwakar Davar, Ryan H. Moy, Lee Ratner, Susan M. Christner, Julianne L. Holleran, Joshua Deppas, Carina Sclafani, John C. Schmitz, Steve Gore, Edward Chu, Christopher J. Bakkenist, Jan H. Beumer, Liza C. Villaruz

Research output: Contribution to journalArticlepeer-review

Abstract

Background: ATR is an apical DDR kinase activated at damaged replication forks. Elimusertib is an oral ATR inhibitor and potentiates irinotecan in human colorectal cancer models. Methods: To establish dose and tolerability of elimusertib with FOLFIRI, a Bayesian Optimal Interval trial design was pursued. Starting elimusertib dose was 20 mg BID days 1, 2, 15 and 16 every 28-day cycle, combined with irinotecan (150 mg/m2) and 5-FU (2000 mg/m2). Results: The trial was stopped after 10 accruals, with four DLT across 4 dose levels including grade 3 febrile neutropenia, mucositis, nausea, vomiting and grade 4 neutropenia. The most common grade 3/4 adverse events were neutropenia, leukopenia, lymphopenia and mucositis. Based on significant toxicities the trial was stopped. PK data for 5-FU and irinotecan were unremarkable and did not account for DLTs. Among the six response evaluable patients, four had stable disease as their best response. Median PFS was 7 months. A first case of ATRi chemotherapy combination related AML (t-AML) was observed. Conclusions: The combination of elimusertib with FOLFIRI was associated with intolerable toxicity. Combination of ATR kinases with chemotherapies that target DNA replication may be associated with significant myelotoxicity. Ongoing ATRi trials should monitor for t-AML. ClinicalTrials.gov

Original languageEnglish
Article number27
JournalCancer Chemotherapy and Pharmacology
Volume95
Issue number1
DOIs
StatePublished - Dec 2025

Keywords

  • ATR inhibition
  • BAY 1895344
  • Elimusertib
  • FOLFIRI
  • Pharmacokinetics

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