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Personalized Therapeutics for K
ATP
-Dependent Pathologies
Colin G. Nichols
Department of Cell Biology & Physiology
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
DBBS - Biochemistry, Biophysics, and Structural Biology
DBBS - Molecular Cell Biology
DBBS - Neurosciences
Institute of Clinical and Translational Sciences (ICTS)
Center for the Investigation of Membrane Excitability Diseases
Hope Center for Neurological Disorders
Research output
:
Contribution to journal
›
Review article
›
peer-review
10
Scopus citations
Overview
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ATP
-Dependent Pathologies'. Together they form a unique fingerprint.
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Keyphrases
SUR1
100%
Personalized Therapy
100%
Effective Treatment
50%
Monogenic
50%
Multiple Tissues
50%
Gain-of-function mutation
50%
Pore-forming Subunit
50%
KATP Channel
50%
Therapeutic Agents
50%
Electrical Activity
50%
ATP-sensitive Potassium Channel
50%
Pharmacology
50%
Selective Channels
50%
Cross-reactivity
50%
Cellular Metabolism
50%
Receptor Subunits
50%
Channel Inhibitor
50%
Inhibitor Drug
50%
Drug-induced
50%
Channel Openers
50%
Armoury
50%
Medicine and Dentistry
Adenosine Triphosphate Sensitive Potassium Channel
100%
Sulfonylurea Receptor
100%
Loss of Function Mutation
50%
Adenosine Triphosphate
50%
Therapeutic Agent
50%
Channel Opener
50%
Cell Metabolism
50%
Receptor Subunit
50%
Cross Reaction
50%
Pharmacology, Toxicology and Pharmaceutical Science
Sulfonylurea Receptor
100%
Adenosine Triphosphate Sensitive Potassium Channel
100%
Adenosine Triphosphate
50%
Isotopes of Potassium
50%
Receptor Subunit
50%
Channel Opener
50%