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Pathobiology of pulmonary fibrosis
E. Crouch
Division of Anatomic and Molecular Pathology (AMP)
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
Institute of Clinical and Translational Sciences (ICTS)
DBBS - Molecular Cell Biology
DBBS - Developmental, Regenerative and Stem Cell Biology
Research output
:
Contribution to journal
›
Review article
›
peer-review
367
Scopus citations
Overview
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Keyphrases
Pathobiology
100%
Pulmonary Fibrosis
100%
Airspace
100%
Lung
40%
Lung Injury
40%
Obliteration
40%
Exudates
40%
Fibroproliferative
40%
Cell Activation
20%
Inflammatory Mediators
20%
Gene Expression
20%
Collagen
20%
Fibrosis
20%
Fibroblasts
20%
Activated State
20%
Inflammatory Cells
20%
Proteoglycan
20%
Macromolecules
20%
Fibronectin
20%
Gas Exchange
20%
Lung Fibrosis
20%
Injury Repair
20%
Spatial Relationship
20%
Connective Tissue Cells
20%
Matrix Components
20%
Interstitium
20%
Resident Cells
20%
Murals
20%
Re-epithelialization
20%
Matrix Deposition
20%
Epithelial Injury
20%
Parenchymal Cells
20%
Structural Remodeling
20%
Alveolar Wall
20%
Matrix Receptor
20%
Fibrous Adhesions
20%
Delayed Repair
20%
Matrix-degrading Enzymes
20%
Alveolar Collapse
20%
Cytoskeletal Alterations
20%
Honeycomb Lung
20%
Medicine and Dentistry
Pulmonary Fibrosis
100%
Lung
100%
Cytokine
33%
Cell Activation
33%
Receptor
33%
Fibrosis
33%
Gene Expression
33%
Inflammatory Cell
33%
Proteoglycan
33%
Inflammatory Mediator
33%
Gas Exchange
33%
Interstitium
33%
Epithelization
33%
Fibronectin
33%
Exudate
33%
Fibroblast
33%
Connective Tissue Cell
33%
Pharmacology, Toxicology and Pharmaceutical Science
Lung Fibrosis
100%
Receptor
33%
Cytokine
33%
Fibrosis
33%
Moroxydine
33%
Fibronectin
33%
Proteoglycan
33%
Exudate
33%