p38 mitogen-activated protein kinase activates peroxisome proliferator-activated receptor α: A potential role in the cardiac metabolic stress response

Keyphrases

• P38 Mitogen-activated Protein Kinase (p38 MAPK) 100%
• Peroxisome Proliferator-activated Receptor 100%
• Metabolic Stress Response 100%
• Myocytes 42%
• Energy Metabolic 42%
• P38 MAP Kinase 14%
• In Vitro Kinase Assay 14%
• Co-transfection 14%
• Transcriptional Repressor 14%
• Receptor Activity 14%
• Transcriptional Level 14%
• Kinase Phosphorylation 14%
• Gene Regulation 14%
• Transient Transfection 14%
• Fatty Acid Oxidation 14%
• Energy Sources 14%
• Response to Stress 14%
• Mammalian Heart 14%
• Energy Production 14%
• Transactivation 14%
• Mitochondrial Biogenesis 14%
• Ligand-dependent 14%
• Receptor Targets 14%
• Nuclear Receptors/peroxisome Proliferator-activated Receptor 14%
• Coactivator 14%
• Downstream Effectors 14%
• PGC-1 14%
• Metabolic Gene Expression 14%
• Hypoxia-ischemia 14%
• Hypertrophic Growth 14%
• Growth Stimuli 14%
• B Domain 14%
• Serine Residues 14%
• Metabolic Genes 14%
• Co-activation 14%
• Stress Stimuli 14%
• Transcription Assay 14%
• Dependent Stress 14%
• CV-1 14%
• Stress-activated Signaling 14%

Biochemistry, Genetics and Molecular Biology

• P38 Mitogen-Activated Protein Kinases 100%
• Peroxisome Proliferator-Activated Receptor 100%
• Kinase 25%
• Phosphotransferase 25%
• Cardiac Muscle Cell 25%
• Gene Control 12%
• Enzyme 12%
• Gene Expression 12%
• Transcription 12%
• Transient Transfection 12%
• Beta Oxidation 12%
• Nuclear Receptor 12%
• Transactivation 12%
• Mitochondrial Biogenesis 12%
• Serine 12%
• Energy Yield 12%

Immunology and Microbiology

• Mitogen 100%
• Peroxisome 100%
• Cardiac Muscle Cell 25%
• Gene Expression 12%
• Muscle Cell 12%
• Transient Transfection 12%
• Gene Control 12%
• Transactivation 12%
• Fatty Acid Oxidation 12%
• Mitochondrial Biogenesis 12%
• Energy Yield 12%