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Old mice lacking high-affinity nicotine receptors resist acoustic trauma
Haiyan Shen
, Zhaoyu Lin
, Debin Lei
, Josiah Han
,
Kevin K. Ohlemiller
, Jianxin Bao
Department of Otolaryngology
Research output
:
Contribution to journal
›
Article
›
peer-review
8
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Scopus citations
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Keyphrases
High Affinity
100%
Acoustic Trauma
100%
Aged Mice
100%
Nicotine Receptors
100%
Noise-induced Hearing Loss
75%
Spiral Ganglion Neurons
50%
Glucocorticoid Signaling
50%
Corticosterone
50%
Knock-in Mouse Model
25%
Signaling Pathway
25%
Inner Hair Cell
25%
Age-related
25%
Organ of Corti
25%
Afferent Fibers
25%
Negative Feedback Loop
25%
Nicotinic Acetylcholine Receptor (nAChR)
25%
Significant Loss
25%
Neural Injury
25%
Synaptic Connectivity
25%
Sword
25%
Cholinergic Transmission
25%
Neuronal Protection
25%
Double-edged
25%
Feedback System
25%
Synthetic Glucocorticoids
25%
Glucocorticoid Drugs
25%
Lateral Olivocochlear Efferents
25%
Neuroscience
Nicotinic Acetylcholine Receptor
100%
Glucocorticoid
100%
Spiral Ganglion Neuron
66%
Corticosterone
66%
Cell Signaling
33%
Cholinergic Transmission
33%
Negative Feedback
33%
Medicine and Dentistry
Noise-Induced Hearing Loss
100%
Nicotinic Receptor
100%
Glucocorticoid
42%
Spiral Ganglion Neuron
28%
Corticosterone
28%
Signal Transduction
14%
Nervous System Injury
14%
Organ of Corti
14%
Inner Hair Cell
14%
Cholinergic Transmission
14%
Negative Feedback
14%
Knockout Mouse
14%