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Ninjurin, a novel adhesion molecule, is induced by nerve injury and promotes axonal growth
Toshiyuki Araki,
Jeffrey Milbrandt
Department of Genetics
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
Institute of Clinical and Translational Sciences (ICTS)
McDonnell Genome Institute (MGI)
Siteman Cancer Center
COVID-19 Researchers
Bursky Center for Human Immunology & Immunotherapy Programs (CHiiPs)
Center of Regenerative Medicine
DBBS - Molecular Cell Biology
DBBS - Developmental, Regenerative and Stem Cell Biology
DBBS - Molecular Genetics and Genomics
DBBS - Neurosciences
Hope Center for Neurological Disorders
Brain Tumor Center
Intellectual and Developmental Disabilities Research Center (IDDRC)
Research output
:
Contribution to journal
›
Article
›
peer-review
202
Scopus citations
Overview
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Keyphrases
Adhesion Molecules
100%
Nerve Injury
100%
Ninjurin1
100%
Axonal Growth
100%
Nerve Regeneration
66%
Schwann Cells
66%
Cell Surface
33%
Epithelial Cells
33%
Dorsal Root Ganglion Neurons
33%
Axonal Degeneration
33%
Phenotypic Change
33%
Axotomy
33%
Neurite Extension
33%
Nerve
33%
Peripheral Nerve Injury
33%
Homophilic Adhesion
33%
Novel Protein
33%
Screening Strategy
33%
Differential Screening
33%
Neuroscience
Cell Adhesion Proteins
100%
Nerve Injury
100%
Nervous System Regeneration
66%
Schwann Cell
66%
In Vitro
33%
Neurite
33%
Dorsal Root Ganglion
33%
Nerve Fiber Degeneration
33%
Epithelial Cells
33%
Peripheral Nerve Injury
33%
Axonal Degeneration
33%