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Neuroprotective mechanisms of heat shock gene expression
B. Joy Snider
Institute of Clinical and Translational Sciences (ICTS)
Hope Center for Neurological Disorders
Research output
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Contribution to journal
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Review article
›
peer-review
Overview
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Keyphrases
Cellular Injury
100%
Cerebral Ischemia
100%
Enhanced Expression
100%
Expressed Protein
100%
Gene Expression
100%
Genetic Manipulation
100%
Glia
100%
Heat Shock Protein Genes
100%
Heat Shock Proteins
100%
Molecular Chaperone
100%
Nervous System
100%
Neurodegenerative Disorders
100%
Neuroprotective Activity
100%
Protein Function
100%
Stress Manipulation
100%
Subcellular Translocation
100%
Therapeutic Target
100%
Thermal Stress
100%
Neuroscience
Brain Ischemia
100%
Cell Damage
100%
Chaperone
100%
Gene Expression
100%
Genetics
100%
Glia
100%
Heat Shock Protein
100%
Nervous System
100%
Neurodegenerative Disorder
100%
Neuroprotective Agent
100%
Protein Function
100%
Biochemistry, Genetics and Molecular Biology
Cell Damage
100%
Chaperone
100%
Gene Expression
100%
Genetic Manipulation
100%
Heat Shock
100%
Heat Shock Protein
100%
Protein Function
100%
Temperature Stress
100%
Immunology and Microbiology
Cell Damage
50%
Chaperone Protein
50%
Gene Expression
100%
Genetic Manipulation
50%
Glia
50%
Heat Shock
100%
Neurological System
50%
Protein Function
50%
Temperature Stress
50%
Therapeutic Target
50%