Skip to main navigation
Skip to search
Skip to main content
WashU Medicine Research Profiles Home
Help & FAQ
Home
Profiles
Departments, Divisions and Centers
Research output
Search by expertise, name or affiliation
Netrin-DCC signaling regulates corpus callosum formation through attraction of pioneering axons and by modulating slit2-mediated repulsion
Thomas Fothergill
, Amber Lee S. Donahoo
, Amelia Douglass
, Oressia Zalucki
, Jiajia Yuan
, Tianzhi Shu
,
Geoffrey J. Goodhill
,
Linda J. Richards
Department of Neuroscience
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
School of Medicine
Department of Developmental Biology
Institute of Clinical and Translational Sciences (ICTS)
DBBS - Computational and Systems Biology
DBBS - Neurosciences
DBBS - Developmental, Regenerative and Stem Cell Biology
Intellectual and Developmental Disabilities Research Center (IDDRC)
DBBS - Molecular Cell Biology
Center of Regenerative Medicine
DBBS - Molecular Genetics and Genomics
DBBS - Cancer Biology
Research output
:
Contribution to journal
›
Article
›
peer-review
82
Scopus citations
Overview
Fingerprint
Fingerprint
Dive into the research topics of 'Netrin-DCC signaling regulates corpus callosum formation through attraction of pioneering axons and by modulating slit2-mediated repulsion'. Together they form a unique fingerprint.
Sort by
Weight
Alphabetically
Keyphrases
Corpus Callosum
100%
Guidance Cues
100%
Repulsion
100%
Commissural Axons
100%
Netrin
100%
Callosal Axons
100%
SLIT2
100%
Pioneering Axons
100%
Axon
50%
Neocortical
50%
Anterior Cingulate Cortex
50%
Forebrain
50%
Nervous System
50%
Spinal Cord
50%
Cortical Function
50%
Left-handed
50%
Evolutionarily Conserved
50%
Right-sided
50%
Callosal
50%
Deleted in Colorectal Cancer
50%
Commissure
50%
Cancer Pathways
50%
Netrin-1
50%
Guidance Mechanism
50%
Neuroscience
Corpus Callosum
100%
Netrin
100%
Axon
100%
Nervous System
14%
Cingulate Cortex
14%
Deleted in Colorectal Cancer
14%
Biochemistry, Genetics and Molecular Biology
Axon
100%
Netrin
100%
Deleted in Colorectal Cancer
14%