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Moving towards therapies for Juvenile Batten disease?
Jonathan D. Cooper
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
Bursky Center for Human Immunology & Immunotherapy Programs (CHiiPs)
Division of Genetics and Genomic Medicine
DBBS - Neurosciences
DBBS - Developmental, Regenerative and Stem Cell Biology
DBBS - Molecular Genetics and Genomics
Research output
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Contribution to journal
›
Comment/debate
19
Scopus citations
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Keyphrases
CLN3
100%
Juvenile Batten Disease
100%
Mouse Model
50%
AMPA Receptor (AMPAR)
50%
Mutant Mice
50%
Batten Disease
50%
Methyl
25%
Glutamate
25%
Overactivation
25%
Receptor Blockade
25%
Transmembrane Protein
25%
Gene-deficient Mice
25%
Disability
25%
Non-competitive
25%
Attenuation
25%
Amino
25%
Effective Therapy
25%
Receptor Activity
25%
Intraperitoneal Injection
25%
Motor Coordination
25%
Disease Phenotype
25%
Neuronal Ceroid Lipofuscinosis
25%
AMPA-type Glutamate Receptor
25%
Motor Skills
25%
Acute Study
25%
Monogenetic
25%
Storage Disorders
25%
Successful Therapy
25%
AMPA Antagonist
25%
Selective Sensitivity
25%
Pharmacology, Toxicology and Pharmaceutical Science
Neuronal Ceroid Lipofuscinosis
100%
Mouse Model
50%
Mouse Mutant
50%
Alpha Amino 3 Hydroxy 5 Methyl 4 Isoxazolepropionic Acid
50%
AMPA Receptor
50%
Diseases
50%
Storage Disease
25%
Glutamic Acid
25%
Glutamate Receptor
25%
Valerian
25%
Lipofuscinosis
25%
Intraperitoneal Injection
25%
Biochemistry, Genetics and Molecular Biology
Batten Disease
100%
Neuronal Ceroid Lipofuscinosis
100%
Mouse Model
40%
Mouse Mutant
40%
AMPA Receptor
40%
Amino Methyl Phosphonic Acid
40%
Motor Performance
20%
Glutamate Receptor
20%
CLN3
20%
Motor Coordination
20%
Receptor Blocking
20%
Glutamic Acid
20%
Neuroscience
Neuronal Ceroid Lipofuscinosis
100%
AMPA Receptor
50%
AMPA
50%
Valerian
25%
Glutamic Acid
25%
Glutamate Receptor
25%
Motor Coordination
25%
Ceroid
25%
Intraperitoneal Injection
25%
Motor Skills
25%