Mitochondrial SIRT3: A new potential therapeutic target for metabolic syndrome

Research output: Contribution to journalShort surveypeer-review

17 Scopus citations

Abstract

In this issue of Molecular Cell, Hirschey et al. demonstrate that loss of the NAD+-dependent deacetylase SIRT3 and resultant mitochondrial protein hyperacetylation play a critical role in the pathogenesis of metabolic syndrome, providing new insights into the therapeutic potential of SIRT3.

Original languageEnglish
Pages (from-to)170-171
Number of pages2
JournalMolecular cell
Volume44
Issue number2
DOIs
StatePublished - Oct 21 2011

Fingerprint Dive into the research topics of 'Mitochondrial SIRT3: A new potential therapeutic target for metabolic syndrome'. Together they form a unique fingerprint.

Cite this