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Metformin inhibits the proliferation of benign prostatic epithelial cells
Zongwei Wang
, Xingyuan Xiao
, Rongbin Ge
, Jijun Li
, Cameron W. Johnson
, Cyrus Rassoulian
, Aria F. Olumi
Division of Anatomic and Molecular Pathology (AMP)
Institute of Clinical and Translational Sciences (ICTS)
Research output
:
Contribution to journal
›
Article
›
peer-review
26
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Scopus citations
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Keyphrases
Metformin
100%
Prostate Epithelial Cells
100%
Benign Prostatic
100%
Benign Prostatic Hyperplasia
80%
Growth Factors
70%
Insulin-like
70%
Insulin-like Growth Factor 1 Receptor (IGF1R)
40%
Cell Proliferation
20%
Cell Population
20%
Epithelial Cells
20%
Fibroblasts
20%
Conditioned Medium
20%
M Cells
20%
3T3 Cells
20%
Diabetes
10%
Prostate
10%
Epithelial Cell Proliferation
10%
Immunocytochemistry
10%
Flow Cytometry
10%
Epidemiologic Studies
10%
Stromal Cells
10%
Culture Media
10%
Paracrine Mechanism
10%
Western Blot
10%
Type 2 Diabetes Prevention
10%
MTS Assay
10%
1 Receptor
10%
Dose-dependent
10%
Antidiabetic Drug
10%
Elderly Men
10%
Medicine and Dentistry
Epithelial Cell
100%
Glycon
100%
Prostate Hypertrophy
80%
Somatomedin C
60%
Receptor
40%
Secretion (Process)
30%
Cell Proliferation
30%
Cell Population
20%
Cell Cycle
20%
3T3 Cells
20%
Microfold Cell
20%
Fibroblast
20%
Stromal Cell
10%
Diabetes
10%
Maturity Onset Diabetes of the Young
10%
Flow Cytometry
10%
Enzyme Linked Immunosorbent Assay
10%
Cohort Effect
10%
Cell Line
10%
Western Blot
10%
Immunocytochemistry
10%
Somatomedin C Receptor
10%
MTS Assay
10%
Anti-Diabetic Medication
10%
Pharmacology, Toxicology and Pharmaceutical Science
Metformin
100%
Prostate Hypertrophy
80%
Somatomedin C
60%
Receptor
40%
Western Blot
10%
Flow Cytometry
10%
Enzyme-Linked Immunosorbent Assay
10%
Somatomedin C Receptor
10%
Anti-Diabetic Medication
10%
Maturity Onset Diabetes of the Young
10%