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Mechanism of Action of N-Butyl Deoxynojirimycin in Inhibiting HIV-1 Infection and Activity in Combination with Nucleoside Analogs
Lee Ratner
, Nancy Vander Heyden
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
Institute of Clinical and Translational Sciences (ICTS)
Siteman Cancer Center
Bursky Center for Human Immunology & Immunotherapy Programs (CHiiPs)
Section of Molecular Oncology & Developmental Therapeutics
Division of Oncology
DBBS - Molecular Microbiology and Microbial Pathogenesis
DBBS - Molecular Genetics and Genomics
DBBS - Cancer Biology
Research output
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Contribution to journal
›
Article
›
peer-review
24
Scopus citations
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Keyphrases
Mechanism of Action
100%
Butyl
100%
Nucleoside Analogues
100%
HIV-1 Infection
100%
1-deoxynojirimycin
100%
Envelope Protein
16%
V3 Loop
16%
Acute Infection
16%
Fusion Activity
16%
Gp120
16%
HIV-1 Envelope
16%
Infection Assay
16%
Infectious Virus Particle
16%
Reverse Transcriptase Activity
16%
CD4 Binding
16%
α-glucosidase Inhibitor
16%
Azidothymidine
16%
Dideoxycytidine
16%
Pharmacology, Toxicology and Pharmaceutical Science
Human Immunodeficiency Virus 1 Infection
100%
1-Deoxynojirimycin
100%
Nucleoside Analog
100%
Infection
16%
HIV
16%
RNA Directed DNA Polymerase
16%
Zidovudine
16%
Glucosidase
16%
Virus Infectivity
16%
Envelope Protein
16%
Zalcitabine
16%
Didanosine
16%