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Loss of Sprouty1 rescues renal agenesis caused by ret mutation
Esteban J. Rozen
, Hagen Schmidt
, Xavier Dolcet
, M. Albert Basson
,
Sanjay Jain
, Mario Encinas
Division of Nephrology
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
Institute of Clinical and Translational Sciences (ICTS)
COVID-19 Researchers
Center of Regenerative Medicine
DBBS - Biomedical Informatics and Data Science
DBBS - Developmental, Regenerative and Stem Cell Biology
DBBS - Molecular Genetics and Genomics
Hope Center for Neurological Disorders
Research output
:
Contribution to journal
›
Article
›
peer-review
43
Scopus citations
Overview
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Keyphrases
Renal Agenesis
100%
Sprouty1
100%
Tyrosine
80%
ERK Pathway
60%
Grb2
40%
Renal Morphogenesis
40%
Binding Site
20%
Knock-in Mouse Model
20%
Kidney
20%
Receptor Tyrosine Kinase
20%
Tumor Suppressor
20%
Adaptor Protein
20%
Negative Signal
20%
Mutant Mice
20%
System Characteristics
20%
Positive Signals
20%
Lethality
20%
Double mutant
20%
Lower Urinary Tract
20%
Enteric Nervous System
20%
Early Postnatal
20%
Effector Proteins
20%
Urinary System
20%
Biochemistry, Genetics and Molecular Biology
Tyrosine
100%
Morphogenesis
50%
Binding Site
25%
Knockout Mouse
25%
Receptor Tyrosine Kinase
25%
Tumor Suppressor Protein
25%
Mouse Mutant
25%
Intestine Innervation
25%