Loss of RBPj in Postnatal Excitatory Neurons Does Not Cause Neurodegeneration or Memory Impairments in Aged Mice

Keyphrases

• Neurodegeneration 100%
• Excitatory Neurons 100%
• Memory Impairment 100%
• Aged Mice 100%
• Age-dependent 60%
• Secretase 60%
• Canonical Notch Signaling 60%
• Gene-deficient Mice 40%
• Mouse Brain 40%
• Learning Impairment 40%
• Notch Receptors 40%
• γ-secretase Inhibitor 40%
• Knock-in Mouse Model 20%
• Alzheimer's Disease 20%
• Forebrain 20%
• Memory Deficits 20%
• Postnatal Mice 20%
• DNA-binding Proteins 20%
• Old Animals 20%
• Specific Substrate 20%
• Notch Signaling 20%
• Cre Driver 20%
• Secretase Activity 20%
• CaMKII 20%
• Brain Pathology 20%
• Untoward Effect 20%
• Receptor Proteolysis 20%
• Olfactory Preference 20%

Neuroscience

• Neurodegeneration 100%
• Memory Disorder 100%
• Secretase 100%
• Notch Signaling Pathway 66%
• Learning Disabilities 33%
• Notch Proteins 33%
• Mouse Brain 33%
• Alzheimer's Disease 16%
• Behavior (Neuroscience) 16%
• DNA-binding Protein 16%
• Proteolysis 16%
• Receptor 16%