Loss of Nkx3.1 leads to the activation of discrete downstream target genes during prostate tumorigenesis

Keyphrases

• Prostate Tumorigenesis 100%
• Downstream Target Genes 100%
• NKX3.1 100%
• C-Myc 33%
• Prostate Cancer 26%
• Pten 26%
• Overexpression 20%
• Mouse Model 13%
• Prostate 13%
• Clusterin 13%
• Cancer Initiation 13%
• Molecular Consequences 13%
• Gene Expression Profiling 6%
• Gene Expression 6%
• Transgenic Mouse Model 6%
• Signaling Pathway 6%
• Gene-deficient Mice 6%
• Transgenic Mice 6%
• Meta-analysis 6%
• Tumor Suppressor Gene 6%
• Early Events 6%
• Tissue Sampling 6%
• Molecular Changes 6%
• Tumor Development 6%
• Gene number 6%
• Constitutively Active 6%
• Transcriptional Regulator 6%
• Atrophy 6%
• Prostate Carcinoma 6%
• Human Prostate 6%
• Epithelial Ingrowth 6%
• Gene Loss 6%
• Tumor Initiation 6%
• Laser Capture Microdissection 6%
• Intraepithelial Neoplasia 6%
• Human Prostate Tissue 6%
• Combined Laser 6%
• Prostate Cancer Model 6%
• AKT Pathway 6%
• Sample Loss 6%
• Prostatic Hyperplasia 6%
• Cancer Gene Expression 6%

Biochemistry, Genetics and Molecular Biology

• Oncogene C Myc 100%
• Carcinogenesis 100%
• Signal Transduction 40%
• Mouse Model 40%
• Clusterin 40%
• Gene Expression 20%
• Gene Expression Profiling 20%
• Transgenic Mouse 20%
• Gene Expression Data 20%
• Tumor Suppressor Gene 20%
• Transgenic Organism 20%
• Gene Loss 20%
• Tumor Initiation 20%
• Laser Capture Microdissection 20%

Pharmacology, Toxicology and Pharmaceutical Science

• Prostate Cancer 100%
• Carcinogenesis 100%
• Neoplasm 60%
• Mouse Model 40%
• Clusterin 40%
• Transgenic Mouse 20%
• Prostate Hypertrophy 20%
• Cancer Model 20%
• Prostate Adenocarcinoma 20%
• Transgenic Organism 20%
• Tumor Suppressor Protein 20%
• Reserpine 20%