Lack of hepatic apoE does not influence early Aβ deposition: Observations from a new APOE knock-in model

Keyphrases

• Knock-in Mouse Model 100%
• Apolipoprotein E 100%
• ApoE 100%
• Amyloid Plaques 36%
• Microglia 27%
• Hepatocytes 27%
• Astrocytes 27%
• Transgenic Mice 18%
• Alzheimer's Disease Pathology 18%
• Gene Expression 9%
• Amyloid Deposition 9%
• Late-onset Alzheimer's Disease 9%
• LoxP 9%
• Lipoprotein Particles 9%
• Type-specific 9%
• Cerebral Amyloid Angiopathy 9%
• Coding Region 9%
• Genetic Risk 9%
• Exon 9%
• Vascular Amyloid 9%
• Gel Analysis 9%
• Isoform-specific 9%
• Biochemical Methods 9%
• Basal Conditions 9%
• Plasma Lipids 9%
• APPPS1 9%
• APP Mice 9%
• Reactive Microglia 9%
• Plasma Lipid Profile 9%
• Amyloid A 9%
• Native Gel 9%
• Histological Techniques 9%
• Primary Cultured Astrocytes 9%
• Dynamic Tool 9%

Neuroscience

• Apolipoprotein E 100%
• Amyloid Protein 63%
• Astrocyte 36%
• Alzheimer's Disease 27%
• Microglia 27%
• Blood Plasma 27%
• Gene Expression 9%
• Cerebral Amyloid Angiopathy 9%
• Exon 9%
• Coding Region 9%
• LoxP Site 9%