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K-Ras
Kian Huat Lim
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
Institute of Clinical and Translational Sciences (ICTS)
Siteman Cancer Center
Bursky Center for Human Immunology and Immunotherapy
Section of Molecular Oncology & Developmental Therapeutics
DBBS - Molecular Cell Biology
DBBS - Immunology
DBBS - Cancer Biology
Research output
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Chapter in Book/Report/Conference proceeding
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Chapter
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peer-review
Overview
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Keyphrases
K-Ras
100%
Mutant KRAS
100%
Cell Proliferation
50%
Signaling Cascades
50%
Mammalian Target of Rapamycin (mTOR)
50%
Clinical Efficacy
50%
Cancer Cells
50%
Improved Survival
50%
Human Cancer
50%
Strategies to Overcome
50%
Genetic Events
50%
Malignant Phenotype
50%
Direct Targeting
50%
Effective Combination
50%
Raf-MEK-ERK
50%
Tolerable Side Effects
50%
ERK Cascade
50%
Resistance Mechanism
50%
Therapeutic Development
50%
Oncogenic Events
50%
Overcoming Resistance
50%
Biochemistry, Genetics and Molecular Biology
Carcinogenesis
100%
Genetics
50%
Cancer Cell
50%
Maturation
50%
Cell Proliferation
50%
Protein Kinase B
50%
Mammalian Target of Rapamycin
50%
Immunology and Microbiology
Maturation
100%
Cancer Cell
100%
Cell Proliferation
100%
Pharmacology, Toxicology and Pharmaceutical Science
Carcinogenesis
100%
Malignant Neoplasm
50%
Protein Kinase B
50%
Side Effect
50%