Intracellular retention of mutant lysyl oxidase leads to aortic dilation in response to increased hemodynamic stress

Keyphrases

• Lysyl Oxidase 100%
• Intracellular Retention 100%
• Hemodynamic Stress 100%
• Aortic Dilation 100%
• Ascending Aorta 60%
• Risk Factors 20%
• Cell number 20%
• Protein Function 20%
• Endoplasmic Reticulum 20%
• Mutant Protein 20%
• Loss Function 20%
• Smooth muscle Cells 20%
• Arterial Wall 20%
• Autophagy 20%
• Isolated Cells 20%
• Golgi 20%
• Aneurysmal Disease 20%
• Descending Aorta 20%
• Thoracic Aortic Aneurysm 20%
• Vessel Wall 20%
• Aortic Diseases 20%
• Ubiquitin-proteasome System 20%
• Inflammatory Cell Infiltration 20%
• Secretion Defect 20%
• Heterozygous Missense mutation 20%
• Thoracic Aortic Dissection 20%
• Arterial Dilatation 20%
• Copper Incorporation 20%
• Wall Structure 20%
• Vascular Aneurysm 20%
• Vessel Dilation 20%

Immunology and Microbiology

• Hemodynamic Stress 100%
• Ascending Aorta 100%
• Secretion (Process) 33%
• Cell Count 33%
• Endoplasmic reticulum 33%
• Smooth Muscle Cell 33%
• Autophagy 33%
• Descending Aorta 33%
• Proteasome 33%
• Inflammatory Cell 33%
• Missense Mutation 33%
• Protein Function 33%

Biochemistry, Genetics and Molecular Biology

• Lysyl Oxidase 100%
• Hemodynamic Stress 100%
• Secretion (Process) 20%
• Protein Function 20%
• Mutant Protein 20%
• Autophagy 20%
• Proteasome 20%
• Missense Mutation 20%
• Cell Count 20%