Intracellular C3 protects human airway epithelial cells from stress-associated cell death

Keyphrases

• Cell Death 100%
• Airway Epithelial Cells 100%
• Inflammatory Cytokines 20%
• Airway 20%
• Lung Tissue 20%
• Inflammation 20%
• Inducer 20%
• Oxidative Stress 20%
• Complement System 20%
• Small Interfering RNA (siRNA) 20%
• Host Defense 20%
• At-risk 20%
• Novel Pathways 20%
• Respiratory Tract 20%
• End-stage Lung Disease 20%
• Free Media 20%
• Environmental Stress 20%
• Starvation 20%
• BAL Fluid 20%
• Host Protection 20%
• CD41 20%
• Tracheobronchial 20%
• Airway Epithelial 20%
• Exogenous Sources 20%
• T Cell Survival 20%
• Intracellular Stores 20%
• CRISPR Knockout 20%
• Defence against Pathogens 20%
• BEAS-2B Cells 20%
• Locally Available 20%
• Pathogen Stress 20%
• Storage Form 20%
• Epithelial Biology 20%
• Endogenous Generation 20%
• PRO-C3 20%
• Environmental Interface 20%

Biochemistry, Genetics and Molecular Biology

• Cell Death 100%
• Anabolism 33%
• T Cell 33%
• Small Interfering RNA 33%
• Liquid 33%
• Oxidative Stress 33%
• Infectious Agent 33%
• Cell Survival 33%
• CD4 33%
• Precursor 33%
• C3b 33%
• Complement System 33%
• Host Resistance 33%
• CRISPR 33%
• Proinflammatory Cytokine 33%
• Environmental Stress 33%

Immunology and Microbiology

• Cell Death 100%
• Airway 100%
• T Cell 14%
• Infectious Agent 14%
• Oxidative Stress 14%
• CD4 14%
• Complement System 14%
• Cell Survival 14%
• Host Resistance 14%
• Proinflammatory Cytokine 14%
• Precursor 14%
• C3b 14%
• Liquid 14%
• Clustered Regularly Interspaced Short Palindromic Repeat 14%
• BEAS-2B Cell Line 14%