Intestinal epithelial HuR modulates distinct pathways of proliferation and apoptosis and attenuates small intestinal and colonic tumor development

Keyphrases

• Apoptosis 100%
• Tumor Development 100%
• Intestinal Epithelial Cells 100%
• Colonic Tumor 100%
• Intestinal Tumor 100%
• Small Intestine 66%
• Tumorigenesis 66%
• Tumor Burden 66%
• Dextran Sulfate Sodium 66%
• Intestinal Tumorigenesis 66%
• Azoxymethane 66%
• Genomic Deletion 33%
• Cell Growth 33%
• Gene Expression 33%
• Transgenic Mouse Model 33%
• Apoptotic Genes 33%
• Cell Proliferation 33%
• Large Intestine 33%
• Proliferating Cells 33%
• Decreased Expression 33%
• Pleiotropic Effects 33%
• Intestinal Cancer 33%
• Pool Size 33%
• Colon Adenocarcinoma 33%
• RNA-binding Protein 33%
• Target RNA 33%
• Villus Height 33%
• Bile Acid Metabolism 33%
• Pro-survival 33%
• Anti-apoptotic 33%
• Pro-apoptotic 33%
• Nucleoplasm 33%
• Colitis-associated Cancer 33%
• Colonic Epithelial Cells 33%
• Pro-apoptotic Factors 33%
• Bile Acid Excretion 33%
• Fecal Bile Acids 33%
• ApcMin Mouse 33%
• Intestinal Growth 33%
• ApcMin 33%
• Asbt 33%
• Acute Gastrointestinal Injury 33%

Pharmacology, Toxicology and Pharmaceutical Science

• Carcinogenesis 100%
• Colon Tumor 100%
• Intestine Tumor 100%
• Neoplasm 50%
• Malignant Neoplasm 50%
• Bile Acid 50%
• Dextran Sulfate 50%
• Azoxymethane 50%
• Antiapoptotic 25%
• Intestine Cancer 25%
• Colon Carcinogenesis 25%
• Colitis 25%
• Transgenic Organism 25%
• RNA Binding Protein 25%
• Intestine Injury 25%

Immunology and Microbiology

• Programmed Cell Death 100%
• Cell Proliferation 66%
• Small Intestine 66%
• Tumor Volume 66%
• Gene Expression 33%
• Gene Deletion 33%
• Pleiotropy 33%
• Cell Growth 33%
• Colitis 33%
• Colon 33%
• Protein RNA Binding 33%
• Transgenic Organism 33%
• Large Intestine 33%
• Bile Acid Metabolism 33%