Inhibition of both the extrinsic and intrinsic death pathways through nonhomotypic death-fold interactions

Young Jae Nam; Kartik Mani; Anthony W. Ashton; Chang Fu Peng; Barath Krishnamurthy; Yukihiro Hayakawa; Peiyee Lee; Stanley J. Korsmeyer; Richard N. Kitsis

doi:10.1016/j.molcel.2021.01.018

Inhibition of both the extrinsic and intrinsic death pathways through nonhomotypic death-fold interactions

Young Jae Nam, Kartik Mani, Anthony W. Ashton, Chang Fu Peng, Barath Krishnamurthy, Yukihiro Hayakawa, Peiyee Lee, Stanley J. Korsmeyer, Richard N. Kitsis

Research output: Contribution to journal › Editorial

1 Scopus citations

Original language	English
Pages (from-to)	638
Number of pages	1
Journal	Molecular cell
Volume	81
Issue number	3
DOIs	https://doi.org/10.1016/j.molcel.2021.01.018
State	Published - Feb 4 2021

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10.1016/j.molcel.2021.01.018

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@article{66b5120e44a14f0284a5edf5c0f0a7c5,

title = "Inhibition of both the extrinsic and intrinsic death pathways through nonhomotypic death-fold interactions",

author = "Nam, \{Young Jae\} and Kartik Mani and Ashton, \{Anthony W.\} and Peng, \{Chang Fu\} and Barath Krishnamurthy and Yukihiro Hayakawa and Peiyee Lee and Korsmeyer, \{Stanley J.\} and Kitsis, \{Richard N.\}",

year = "2021",

month = feb,

day = "4",

doi = "10.1016/j.molcel.2021.01.018",

language = "English",

volume = "81",

pages = "638",

journal = "Molecular cell",

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TY - JOUR

T1 - Inhibition of both the extrinsic and intrinsic death pathways through nonhomotypic death-fold interactions

AU - Nam, Young Jae

AU - Mani, Kartik

AU - Ashton, Anthony W.

AU - Peng, Chang Fu

AU - Krishnamurthy, Barath

AU - Hayakawa, Yukihiro

AU - Lee, Peiyee

AU - Korsmeyer, Stanley J.

AU - Kitsis, Richard N.

PY - 2021/2/4

Y1 - 2021/2/4

UR - https://www.scopus.com/pages/publications/85100983408

U2 - 10.1016/j.molcel.2021.01.018

DO - 10.1016/j.molcel.2021.01.018

M3 - Editorial

C2 - 33545060

AN - SCOPUS:85100983408

SN - 1097-2765

VL - 81

SP - 638

JO - Molecular cell

JF - Molecular cell

IS - 3

ER -

Inhibition of both the extrinsic and intrinsic death pathways through nonhomotypic death-fold interactions

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