Improved control of tuberculosis and activation of macrophages in mice lacking protein kinase R

Keyphrases

• Tuberculosis 100%
• Mycobacterium Tuberculosis (M. tb) 100%
• Macrophage Activation 100%
• Improved Control 100%
• Protein Kinase R 100%
• Macrophages 66%
• Interferon-γ 66%
• Double-stranded RNA 33%
• Wild Mice 33%
• Apoptosis 33%
• Protective Effect 33%
• Gene-deficient Mice 33%
• Host Factors 33%
• Leading Causes of Death 33%
• Bacterial Infection 33%
• Interleukin-10 33%
• Mammalian Host 33%
• Adjunctive Therapy 33%
• Protein Kinase PKR 33%
• Microbial Pathogens 33%
• Pulmonary Pathology 33%
• Restraining Effect 33%
• PKR Inhibitor 33%

Pharmacology, Toxicology and Pharmaceutical Science

• Mycobacterium Tuberculosis 100%
• Protein Kinase R 100%
• Infection 33%
• Wild Type Mouse 33%
• Infectious Agent 33%
• Bacterial Infection 33%
• Interleukin 10 33%
• Host Factor 33%

Immunology and Microbiology

• Macrophage 100%
• Mycobacterium Tuberculosis 75%
• Wild Type Mouse 25%
• Infectious Agent 25%
• Interleukin 10 25%
• Host Factor 25%
• Macrophage Activation 25%
• Programmed Cell Death 25%