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Immune-Mediated Glomerular Injury
J. P. Gaut
Institute of Clinical and Translational Sciences (ICTS)
Department of Pathology & Immunology
Division of Anatomic and Molecular Pathology (AMP)
Research output
:
Chapter in Book/Report/Conference proceeding
›
Chapter
›
peer-review
1
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Scopus citations
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Keyphrases
Immune Response
100%
Glomerular Injury
100%
Lupus Nephritis
66%
Membranous Glomerulonephritis
66%
Postinfectious Glomerulonephritis
66%
Disease Progression
33%
Inflammatory Mediators
33%
Genetic Studies
33%
Immune Complex
33%
Prevention Strategies
33%
Disease Susceptibility
33%
Glomerular Disease
33%
Therapeutic Strategies
33%
Pathophysiological Mechanisms
33%
Renal Biopsy
33%
Disease Entity
33%
Inflammatory Cell Infiltration
33%
Complement System Activation
33%
Immune Complex Diseases
33%
Medicine and Dentistry
Glomerulonephritis
100%
Lupus Nephritis
100%
Membranous Glomerulonephritis
100%
Diseases
100%
Disease Predisposition
50%
Disease Exacerbation
50%
Complement System
50%
Inflammatory Cell
50%
Antigen Antibody Complex
50%
Glomerulopathy
50%
Inflammatory Mediator
50%
Renal Biopsy
50%
Immune Complex Disease
50%
Immunology and Microbiology
Glomerulonephritis
100%
Lupus Erythematosus
100%
Nephritis
100%
Membranous Glomerulonephritis
100%
Mediator
50%
Complement System
50%
Immune Complex
50%
Inflammatory Cell
50%
Immune Complex Disease
50%
Susceptibility
50%