Hypothesis: Antigen-specific T cells prime central nervous system endothelium for recruitment of nonspecific inflammatory cells to effect autoimmune demyelination

A. H. Cross; B. Cannella; C. F. Brosnan; C. S. Raine

doi:10.1016/0165-5728(91)90111-J

Hypothesis: Antigen-specific T cells prime central nervous system endothelium for recruitment of nonspecific inflammatory cells to effect autoimmune demyelination

A. H. Cross, B. Cannella, C. F. Brosnan, C. S. Raine

Research output: Contribution to journal › Article › peer-review

Original language	English
Pages (from-to)	237-244
Number of pages	8
Journal	Journal of Neuroimmunology
Volume	33
Issue number	3
DOIs	https://doi.org/10.1016/0165-5728(91)90111-J
State	Published - Sep 1991

Keywords

Adhesion
Autoimmunity
Central nervous system
Multiple sclerosis
T cell

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10.1016/0165-5728(91)90111-J

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@article{6e1fc00ad1794104b7c2937157c8454e,

title = "Hypothesis: Antigen-specific T cells prime central nervous system endothelium for recruitment of nonspecific inflammatory cells to effect autoimmune demyelination",

keywords = "Adhesion, Autoimmunity, Central nervous system, Multiple sclerosis, T cell",

author = "Cross, {A. H.} and B. Cannella and Brosnan, {C. F.} and Raine, {C. S.}",

note = "Funding Information: The authors thank Drs. Krzysztof Selmaj and Giovanni Monastra for helpful discussion, and Howard Finch, Tom O'Mara, Miriam Pakingan and Earl Swanson for excellent technical assistance. Michele Briggs assisted in the preparation of the manuscript. This work was supported in part by grants JF 2042-A-2, RG 1001-G-7 and RG 1089 from the National Multiple Sclerosis Society; and NS 11920, NS 08952 and NS 07098 from the National Institutes of Health. A.H.C. is a Harry Weaver Neuroscience Scholar of the National Multiple Sclerosis Society.",

year = "1991",

month = sep,

doi = "10.1016/0165-5728(91)90111-J",

language = "English",

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pages = "237--244",

journal = "Journal of Neuroimmunology",

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T2 - Antigen-specific T cells prime central nervous system endothelium for recruitment of nonspecific inflammatory cells to effect autoimmune demyelination

AU - Cross, A. H.

AU - Cannella, B.

AU - Brosnan, C. F.

AU - Raine, C. S.

N1 - Funding Information: The authors thank Drs. Krzysztof Selmaj and Giovanni Monastra for helpful discussion, and Howard Finch, Tom O'Mara, Miriam Pakingan and Earl Swanson for excellent technical assistance. Michele Briggs assisted in the preparation of the manuscript. This work was supported in part by grants JF 2042-A-2, RG 1001-G-7 and RG 1089 from the National Multiple Sclerosis Society; and NS 11920, NS 08952 and NS 07098 from the National Institutes of Health. A.H.C. is a Harry Weaver Neuroscience Scholar of the National Multiple Sclerosis Society.

PY - 1991/9

Y1 - 1991/9

KW - Adhesion

KW - Autoimmunity

KW - Central nervous system

KW - Multiple sclerosis

KW - T cell

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DO - 10.1016/0165-5728(91)90111-J

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AN - SCOPUS:0025895393

SN - 0165-5728

VL - 33

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EP - 244

JO - Journal of Neuroimmunology

JF - Journal of Neuroimmunology

IS - 3

ER -

Hypothesis: Antigen-specific T cells prime central nervous system endothelium for recruitment of nonspecific inflammatory cells to effect autoimmune demyelination

Keywords

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